By Hardy G. H., Littlewood J. E.
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Extra resources for Some Problems of Diophantine Approximation A Remarkable Trigonometrical Series (1916)(en)(4s)
For instance, Chien et al. 5”C. Duncker et al. (1996) showed an even steeper re- Myocardial Protection by Brief Ischemic and NonischemicStress 35 lationship between body core temperature and infarct size produced by a 45-minute CAO and 4 hours of reperfusion in swine, as 20% of the area at risk became infarcted with a 1°C increase in temperature in the range of 35°C to 39°C. All three species (rat, rabbit, and pig) lack a significant coronary collateral circulation and this determinant of infarct size can thus be excluded as a possible explanation.
39) 270, HI 189-HI199. C. (1996). Metabolic adaptation during a sequence of no-flow and low-flow ischemia. Circulation 94, 2587-2596. , and Solares,J. (1988). Influenceof tachycardia and arterial hypertension on infarct size in the pig. Cardiovasc. Res. 22,620-626. D. (1996). Myocardial protection by brief ischemia in noncardiac tissue. Circulation 94,2193-2200. D. (1994). Cardioprotection by transient renal ischemia, an in vivo study in rats. ]. D. (1995). Myocardial protection by transient renal ischemia in rats is temperature dependent.
1994b) demonstrated that the delayed protective effects of cardiac pacing were not seen in dogs pretreated with dexamethasone, suggesting the possibility of the induction of cyclo-oxygenase I1 or nitric oxide synthase I1 by cardiac pacing. There are, however, a number of other possible explanations for the detrimental effects of dexamethasone. The second study comes from the use of icatibant to block bradykinin B, receptors. These results suggest that there may be similar mechanisms for the early and delayed antiarrhythmiceffects of ischemic preconditioning and cardiac pacing.
Some Problems of Diophantine Approximation A Remarkable Trigonometrical Series (1916)(en)(4s) by Hardy G. H., Littlewood J. E.